An Unbiased View of CFSE
11-thirteen,29 The unique activity of HDAC inhibitors for AITL is likely to be stated by the fact that epigenetic dysregulation plays a critical part in AITL pathogenesis. Meanwhile, modern molecular research unveiled that AITL and nodal PTCL with T-follicular helper cell (TFH) phenotype share many of the recurrent genetic alterations in epigenetic